(1) Continuity of chronic superficial gastritis: Chronic atrophic gastritis can develop from chronic superficial gastritis. After 5-8 years of follow-up, 6 hospitals, including PLA General Hospital, reported 64 cases of superficial gastritis/KLOC-0, of which 34 cases turned into chronic atrophic gastritis (20.7%). The etiology of chronic superficial gastritis can be the etiology and stress of chronic atrophic gastritis.
(2) Genetic factors: According to the observation of Varis, the incidence of chronic atrophic gastritis in the first generation of patients with chronic atrophic gastritis increased significantly, and the genetic factors of malignant blood deficiency were also significant. The incidence of relatives is 20 times higher than that of the control group, indicating that chronic atrophic gastritis is probably related to genetic factors.
(3) Metal War: The incidence of gastric ulcer and atrophic gastritis among workers exposed to lead is high. In addition to lead, many heavy metals, such as mercury, tellurium, copper and zinc, have certain damage to gastric mucosa.
(4) Radiation: Radiation therapy for ulcers or other tumors will damage or even shrink the gastric mucosa.
(5) Blood iron deficiency: Many explanations of blood iron deficiency are closely related to atrophic gastritis. Badanoch reported 50 cases of blood iron deficiency, with normal gastric mucosa, superficial gastritis and atrophic gastritis accounting for 65,438 0.4%, 46% and 40% respectively. However, the mechanism of gastritis caused by blood deficiency is still unclear. Some scholars believe that gastritis is the primary disease, because the gastric acid in gastritis is too low to absorb iron, or it becomes blood deficiency due to gastric bleeding; Another view is that there is blood deficiency first, because iron deficiency in the body affects the renewal speed of gastric mucosa and is prone to inflammation.
(6) Biological factors: The effects of chronic epidemics such as hepatitis and tuberculosis on the gastrointestinal tract have also attracted people's attention in detail. Patients with chronic liver disease often have symptoms and signs of chronic gastritis, and gastric mucosa staining also shows that there are hepatitis B virus antigen-antibody complexes in the gastric mucosa of patients with hepatitis B. Ruijin Hospital reported 9 cases of atrophic gastritis, including 24 cases of chronic hepatitis (26.4%). Therefore, the influence of chronic epidemic diseases, especially chronic liver diseases, on the stomach deserves to be elaborated.
(7) Physical factors: Clinical statistical results show that the incidence of this disease is obviously positively correlated with age. The older you get, the worse the "resistance" of gastric mucosa, which is vulnerable to external unfavorable factors.
(8) Bile or duodenal juice reflux: Due to postoperative dysfunction of pyloric sphincter or gastrojejunum anastomosis, bile or duodenal juice can reflux into the stomach, destroying the gastric mucosal barrier and stimulating the gastric wall. +and pepsin diffuse back to mucosa, causing a series of pathological changes, leading to chronic superficial gastritis, which can develop into chronic atrophic gastritis.
(9) Immune elements: parietal cell antibodies or endogenous factor antibodies can often be found in the blood and gastric juice of patients with atrophic gastritis, especially the plasma cells of atrophic mucosa, so it is considered that autoimmune reaction is the related cause of chronic atrophic gastritis. In recent years, it has been found that a few patients with gastric antrum gastritis have gastrin-secreting cell antibodies, which are special autoimmune antibodies of cells and belong to Ig.
G series. The changes of lymphocytes in vitro and the inhibition of leukocyte movement in patients with atrophic gastritis are very different, suggesting that cellular immune response is also of great significance in the production of atrophic gastritis.
(10) Helicobacter pylori (HP) infection: 1983, Australian scholars Marshall and Warren scattered HP from gastric antrum mucosa and epithelial cells of patients with chronic gastritis for the first time. After that, many scholars discussed the patients with chronic gastritis, and HP was produced in the gastric mucosa of 60% ~ 90% patients with chronic gastritis, and then it was found that the exposure level of HP was positively correlated with the inflammation level of gastric mucosa. At the eighth meeting of 1986 World Gastroenterology Society, it was suggested that HP infection was one of the neurological causes of chronic gastritis.
In addition; Such as improper diet, long-term alcohol and tobacco addiction, drug abuse, chronic inflammation of upper respiratory tract, dysfunction of central nervous system, damage to gastric mucosa, resection of gastric antrum secreting gastrin after subtotal gastrectomy, resulting in gastric mucosal malnutrition. All these can easily lead to gastric mucosal damage, atrophy and inflammatory changes.
As early as 1973, Strickland et al. divided atrophic gastritis into two independent types: type A and type B according to the serum immunological examination and the distribution of gastric mucosal lesions. Type A atrophic gastritis is mainly found in the body of the stomach, with diffuse distribution, normal gastric antrum mucosa, positive serum parietal cell antibody, increased serum gastrin, decreased or lack of gastric acid and endogenous factors, and prone to pernicious anemia, also known as autoimmune gastritis. Type B atrophic gastritis is more common in the antrum of stomach, showing multifocal distribution. Serum parietal cell antibody is negative, serum gastrin is normal, gastric acid secretion is normal or slightly decreased, and there is no pernicious anemia, which is easy to be complicated with gastric cancer. This is simple atrophic gastritis. Since then, Glass called atrophic gastritis involving both gastric antrum and gastric body AB type.
In China, according to Strickland classification, type B atrophic gastritis is common, while type A atrophic gastritis is rare. Some patients with atrophic gastritis have both gastric antrum inflammation and parietal cell antibody, which cannot be classified into the above two types. Therefore, many domestic scholars have put forward a classification method suitable for China's specific situation, and classified chronic atrophic gastritis as A? Type 1, a? Type 2, type b 1 and b? Type 2. Its typing is mainly based on the situation of autoantibodies. Serum parietal cell antibody positive belongs to type A, while serum parietal cell antibody negative belongs to type B. Type A is divided into two subtypes, and the one without gastric antrum lesion is A? 1 type, patients with gastric antrum and body lesions are a? Type 2. Type B is divided into type B according to the severity of gastric body and antrum lesions. 1 type (gastric antrum lesion is heavier than stomach) and b? Type 2 (gastric body lesions are heavier than gastric antrum lesions or gastric body antrum lesions are similar) two subtypes.
In short, there is no completely unified classification method for chronic atrophic gastritis at present. People are used to classifying chronic atrophic gastritis into type A and type B according to Strickland classification, and there is no specific clinical manifestation. Therefore, the diagnosis of chronic atrophic gastritis needs clinical manifestations combined with relevant auxiliary examinations, especially gastroscopy and gastric mucosal biopsy. The following systematically introduces the diagnosis basis of chronic atrophic gastritis:
(1) Clinical manifestations: mainly anorexia, nausea, belching, abdominal fullness or dull pain, and a few patients may have upper gastrointestinal bleeding, emaciation, anemia, brittle nails, glossitis or atrophy of tongue papilla.
(2) Laboratory inspection
① Analysis of gastric juice: Most patients with type A CAG have no acid or low acid, while patients with type B CAG may have normal or low acid.
② Determination of pepsinogen: Pepsinogen is secreted by main cells, and the content of pepsinogen in blood and urine of chronic atrophic gastritis is decreased.
③ Determination of serum gastrin: G cells in gastric antrum mucosa secrete gastrin. In patients with type A CAG, serum gastrin is often significantly increased; The atrophy of gastric antrum mucosa in patients with type B CAG directly affects the secretion of gastrin by G cells, and the serum gastrin is lower than normal.
④ Immunological examination: The determination of parietal cell antibody (PCA), intrinsic factor antibody (IFA) and gastrin-secreting cell antibody (GCA) can be used as an auxiliary diagnosis of chronic atrophic gastritis and its classification.
(3)X-ray examination: X-ray barium meal examination showed that most patients with atrophic gastritis had no abnormal findings. The double contrast examination of gas and barium can show that the gastric mucosal folds are flat and thin, the serrated gastric mucosal folds become thinner or disappear, the gastric fundus is smooth, and some gastric mucosa with antritis can be serrated or rough.
(4) Gastroscope and biopsy: Gastroscope and biopsy are the most reliable diagnostic methods. Endoscopic diagnosis should include lesion site, degree of atrophy, degree of intestinal metaplasia and atypical hyperplasia. Visually, the mucosa of atrophic gastritis is mostly pale or gray with thin or flat folds. Mucosa can be red and white, and in severe cases, there are scattered white patches. Submucosal vessels show atrophic gastritis, red reticular arterioles or capillaries can be seen, severe atrophic gastritis can be seen, and epithelial cells can proliferate to form small particles or larger nodules. And mucosal erosion and bleeding. Pathology of gastric mucosa biopsy mainly showed that glands atrophied and disappeared in different degrees, replaced by pyloric glands or intestinal glands metaplasia, and interstitial inflammation infiltration was significant.
Chronic atrophic gastritis has no specific clinical manifestations, and the diagnosis of chronic atrophic gastritis needs clinical manifestations combined with related auxiliary examinations, especially gastroscopy and gastric mucosal biopsy. The following systematically introduces the diagnosis basis of chronic atrophic gastritis:
The clinical manifestations of chronic atrophic gastritis are not only lack of specificity, but also not completely consistent with the degree of lesions. Clinically, some patients with chronic atrophic gastritis may have no obvious symptoms. However, most patients may have burning pain, distending pain, dull pain or fullness, especially after eating, loss of appetite, nausea, belching, constipation or diarrhea. In severe cases, emaciation, anemia, brittle nails, glossitis or atrophy of tongue papilla may occur, and a few cases of gastric mucosal erosion may be accompanied by upper gastrointestinal bleeding. Among them, type A atrophic gastritis complicated with pernicious anemia is rare in China. The disease has no specific signs, and the upper abdomen may have mild tenderness.
Chronic atrophic gastritis is a common stomach disease. Arteriosclerosis, insufficient gastric blood flow, tobacco, alcohol and tea hobbies are all easy to destroy the barrier function of gastric mucosa and cause chronic atrophic gastritis. In atrophic gastritis, the gastric mucosa atrophies and is replaced by intestinal epithelial cells, that is, intestinal metaplasia; If inflammation continues to develop, the cell growth is atypical, that is, anaplasia; Even cell proliferation leads to cancer. The clinical manifestations are only indigestion symptoms such as fullness in the upper abdomen, belching and loss of appetite. Sometimes, due to the destruction of stomach factors, vitamin B 12 malabsorption will cause anemia. Endoscopy and biopsy are the only means to diagnose the disease. Atrophic gastritis can be treated with drugs, and it should be checked regularly to prevent cancer.
Matters needing attention
1. Eat fresh and nutritious food to ensure adequate intake of protein, vitamins and iron. Eat on time, don't overeat, don't eat cold or overheated food, and don't use or use less pungent condiments such as fresh and spicy powder.
2. Restrain drinking, do not smoke, and avoid the harm of nicotine to gastric mucosa; Avoid taking anti-inflammatory painkillers for a long time, such as aspirin and corticosteroids, to reduce gastric mucosal damage.
3. Check regularly and do gastroscopy if necessary.
4. If symptoms worsen, emaciation, anorexia, black stool, etc. You should go to the hospital for examination in time.
First, general treatment: try to remove all pathogenic factors, develop good eating habits, avoid foods and drinks that are irritating to gastric mucosa, and quit smoking and drinking. Avoid taking drugs harmful to gastric mucosa;
Second, improve the acidity of gastric cavity: people with low gastric acid can be given 10% dilute hydrochloric acid. Some people advocate intramuscular injection of pentagastrin to promote gastric acid secretion. Try to avoid using drugs that reduce gastric acid;
Third, protect the gastric mucosa: strengthening the gastric mucosal barrier and avoiding mucosal damage are particularly important for the treatment of atrophic gastritis. Can be given sucralfate, colloidal bismuth, prostaglandin E (Hick's ulcer), Schweitzer and other drugs. Taking vitamins for a long time may have a positive effect on mucosal protection.
Fourth, eliminate helicobacter pylori: for chronic atrophic gastritis with positive helicobacter pylori, we should actively eliminate helicobacter pylori and often use antibiotics or colloidal bismuth;
Fifth, enhance gastric emptying ability: gastrointestinal motility drugs can be used to accelerate gastric emptying and reduce the damage of bile to gastric mucosa, and motilium or cisapride can be used. For example, (1) metoclopramide; (2) motilium; (3) Sisapri; Fourth, cholestyramine. The dosage and usage of the above drugs should be in accordance with the doctor's advice.
At present, it is believed that if chronic atrophic gastritis is found early and treated actively in time, the atrophic glands at the lesion site can be recovered and transformed into superficial gastritis or cured, which has changed people's previous understanding of the irreversibility of chronic atrophic gastritis.
Chronic atrophic gastritis is known as the background disease of gastric cancer, but according to the analysis of domestic follow-up data for many years, most of them have a good prognosis, and a few of them can become cancerous, with the canceration rate of 1% ~ 3%. Simple atrophic gastritis, especially mild and moderate atrophic gastritis, has a low canceration rate, while patients with severe atrophic gastritis with moderate and severe intestinal metaplasia and severe atypical hyperplasia, or with carcinoembryonic antigen, have a high canceration rate, which should be paid great attention to, and the gastroscope should be reviewed every 3 ~ 6 months. If possible, the cellular deoxyribonucleic acid (DNA) content and tumor-associated antigen can be checked. Postoperative atrophic gastritis patients have a high canceration rate due to the long-term stimulation of bile reflux. Active measures should be taken to reduce the stimulation of alkaline reflux and prevent cancer.
We must first know what atrophic gastritis is. Atrophic gastritis refers to the atrophy or disappearance of glands located in gastric mucosa due to inflammatory infiltration and other reasons. With the increase of age, its incidence rate increased obviously. The main manifestations are fullness, discomfort or pain in the upper abdomen, which is obvious after meals, accompanied by belching, acid reflux, nausea, vomiting, loss of appetite and other dyspeptic symptoms.
It is reported that the incidence of gastric cancer in atrophic gastritis is about 10%. Animal experiments and clinical observation show that the process of gastritis evolving into gastric cancer is from superficial gastritis to atrophic gastritis to intestinal metaplasia and atypical hyperplasia to gastric cancer.
It can be seen that it is a long process from atrophic gastritis to gastric cancer, in which intestinal metaplasia and atypical hyperplasia are the bridges between disease and gastric cancer. Gastric mucosa is replaced by intestinal mucosa, which is called intestinal metaplasia of gastric mucosa Its enzyme system is not perfect, which makes the absorbed carcinogens accumulate locally, leading to atypical proliferation and mutation into cancer.
The long-term follow-up observation of atrophic gastritis by scholars at home and abroad shows that the incidence of gastric cancer is related to the length of the history of atrophic gastritis and the severity of the disease, but such lesions are reversible. So atrophic gastritis is not equal to gastric cancer.
How to make atrophic gastritis patients develop in a good direction and stay away from gastric cancer? Here are some precautions and simple and effective treatment methods.
Facing up to the illness and excluding psychological factors can lead to cerebral cortex dysfunction, which will have an impact on gastric juice secretion, mucosal vascular filling degree and gastric wall peristalsis. You must learn to adjust your emotions and live a relaxed and happy life.
Regular diet, light diet should not be too full, too hungry, too cold, too hot, too hard, too greasy. Eat less pickled products and spicy food. Smoking, drinking, strong tea, coffee and other bad hobbies. There are many drugs to treat atrophic gastritis according to the doctor's advice, such as multi-enzyme tablets, zinc glycyrrhizinate, weifukang and so on. It should be taken under the guidance of a doctor. Pay attention to regular follow-up visits and review gastroscopy. Carefully use or not use aspirin, prednisone, indomethacin, painkillers and other drugs that are easy to hurt the stomach. There is an effective prescription for atrophic gastritis, which is to mix a spoonful of royal jelly and a spoonful of honey with a cup of warm water, and drink it on an empty stomach every morning, which is beneficial and harmless for a long time.
Atrophic gastritis is accompanied by intestinal metaplasia and atypical hyperplasia. It is necessary to eat more foods rich in β-carotene, vitamin C and folic acid on weekdays, such as kiwi fruit, citrus, strawberry, animal liver and green vegetables, which can improve the condition. Severe intestinal metaplasia and atypical hyperplasia (also called precancerous lesions) should be paid great attention to and followed up regularly. If necessary, the focus should be surgically removed in time to cut off the source of the disease.
Chronic gastritis (superficial gastritis and atrophic gastritis) is a common and frequently-occurring disease. According to the gastroscopy survey, the incidence of chronic gastritis in China is as high as 60%, and atrophic gastritis accounts for about 1/5.
It used to be thought that atrophic gastritis was a prelude to gastric cancer (precancerous lesion), but now it seems that this understanding is biased. But atrophic gastritis has a certain relationship with gastric cancer. The basis is as follows: ① Studies have proved that patients with two kinds of gastric mucosal lesions, "colonic metaplasia" and "atypical hyperplasia", may develop gastric cancer. This is an accepted fact. ② According to epidemiological investigation, the incidence of atrophic gastritis is high in the population with high incidence of gastric cancer. ③ Pathological examination showed that atrophic lesions of mucosa around gastric cancer were common.
It can be said that atrophic gastritis is not a precancerous lesion, but if it is allowed to develop naturally, a few cases may develop into gastric cancer. Therefore, we must take measures to treat it seriously, so as to keep the condition stable (it is difficult to cure it completely) and avoid the occurrence of cancer. Preventive measures for canceration of atrophic gastritis (for canceration, treatment also belongs to prevention) mainly include:
1. Antibacterial therapy. Now the medical community recognizes that Helicobacter pylori (HP) is definitely the pathogen of chronic gastritis, so antibacterial treatment should be carried out first.
2. Oral gastric mucosal protective agent. Commonly used drugs are: sucralfate, which can combine with mucin in gastric mucosa to form a protective film to protect gastric mucosa; Gastrodin can form a film in the stomach to cover the mucosal surface, reducing the stimulation of bile reflux on the gastric mucosa; Chlorophyll can promote inflammation and protect gastric mucosa; Hericium erinaceus tablets can protect gastric mucosa.
3. Increase the concentration of gastric acid. Atrophic gastritis is often acid-free or acid-deficient (gastric cancer is acid-free), and pepsin mixture or dilute hydrochloric acid mixture can be used; Low dose intramuscular injection of pentagastrin can nourish and protect gastric mucosa and promote the secretion of hydrochloric acid by parietal cells.
4. Take vitamins. It can improve human immunity, enhance the activity of detoxification enzymes in human body, inhibit the growth of cancer cells and prevent abnormal cell metabolism.
5. Treat bile reflux. After pyloric sphincter dysfunction or gastrojejunostomy, the gastric mucosal barrier can be destroyed due to long-term bile reflux, leading to chronic superficial gastritis, and then develop into chronic atrophic gastritis. In this case, gastric motility drugs can be used to prevent bile reflux, so as to protect gastric mucosa.
6. Diet therapy. Those with low gastric acid and bile reflux should eat more high-protein and low-fat diets such as lean meat, poultry meat, fish and milk. You should chew slowly and avoid overeating; Avoid drinking strong tea, spirits (especially alcoholism), coffee and eating spicy, overheated and rough food for a long time.
7. Eliminate some pathogenic factors, such as quitting smoking, and avoid taking drugs that stimulate gastric mucosa for a long time (such as sodium salicylate, indomethacin, phenylbutazone and aspirin). ), relieve mental stress, maintain emotional optimism, thereby improving immune function and enhancing disease resistance.
8. Review regularly. Patients with atrophic gastritis with incomplete colonic metaplasia and atypical hyperplasia should have regular gastroscopy: general atrophic gastritis for 3 years 1 time, incomplete colonic metaplasia with mild atypical hyperplasia for 0 years 1 time, moderate atypical hyperplasia for 3 months 1 time and severe atypical hyperplasia (canceration rate 1 time).
We must first know what atrophic gastritis is. Atrophic gastritis refers to the atrophy or disappearance of glands located in gastric mucosa due to inflammatory infiltration and other reasons. With the increase of age, its incidence rate increased obviously. The main manifestations are fullness, discomfort or pain in the upper abdomen, which is obvious after meals, accompanied by belching, acid reflux, nausea, vomiting, loss of appetite and other dyspeptic symptoms.
It is reported that the incidence of gastric cancer in atrophic gastritis is about 10%. Animal experiments and clinical observation show that the process of gastritis evolving into gastric cancer is from superficial gastritis to atrophic gastritis to intestinal metaplasia and atypical hyperplasia to gastric cancer.
It can be seen that it is a long process from atrophic gastritis to gastric cancer, in which intestinal metaplasia and atypical hyperplasia are the bridges between disease and gastric cancer. Gastric mucosa is replaced by intestinal mucosa, which is called intestinal metaplasia of gastric mucosa Its enzyme system is not perfect, which makes the absorbed carcinogens accumulate locally, leading to atypical proliferation and mutation into cancer.
The long-term follow-up observation of atrophic gastritis by scholars at home and abroad shows that the incidence of gastric cancer is related to the length of the history of atrophic gastritis and the severity of the disease, but such lesions are reversible. So atrophic gastritis is not equal to gastric cancer.
How to make atrophic gastritis patients develop in a good direction and stay away from gastric cancer? Here are some precautions and simple and effective treatment methods.
Facing up to the illness and excluding psychological factors can lead to cerebral cortex dysfunction, which will have an impact on gastric juice secretion, mucosal vascular filling degree and gastric wall peristalsis. You must learn to adjust your emotions and live a relaxed and happy life.
Regular diet, light diet should not be too full, too hungry, too cold, too hot, too hard, too greasy. Eat less pickled products and spicy food. Smoking, drinking, strong tea, coffee and other bad hobbies. There are many drugs to treat atrophic gastritis according to the doctor's advice, such as multi-enzyme tablets, zinc glycyrrhizinate, weifukang and so on. It should be taken under the guidance of a doctor. Pay attention to regular follow-up visits and review gastroscopy. Carefully use or not use aspirin, prednisone, indomethacin, painkillers and other drugs that are easy to hurt the stomach. There is an effective prescription for atrophic gastritis, which is to mix a spoonful of royal jelly and a spoonful of honey with a cup of warm water, and drink it on an empty stomach every morning, which is beneficial and harmless for a long time.
Atrophic gastritis is accompanied by intestinal metaplasia and atypical hyperplasia. It is necessary to eat more foods rich in β-carotene, vitamin C and folic acid on weekdays, such as kiwi fruit, citrus, strawberry, animal liver and green vegetables, which can improve the condition. Severe intestinal metaplasia and atypical hyperplasia (also called precancerous lesions) should be paid great attention to and followed up regularly. If necessary, the focus should be surgically removed in time to cut off the source of the disease.
Chinese medicine believes that people's mental factors, such as seven internal injuries, great joy and great sorrow, anxiety, fear, long-term mental tension, wind, cold, heat, dampness, dryness, fire, etc., can all hurt the spleen and stomach, leading to gastritis.
Because the histological changes of gastric mucosa last for a long time, and the relationship between atrophic gastritis and gastric cancer is very close, for some cases, gastric cancer can occur if the treatment is not timely and inappropriate.
Western medicine has no special treatment and very effective methods for atrophic gastritis, only symptomatic treatment, pain relief, digestion aid, bile reflux reduction and antibacterial treatment.
Traditional Chinese medicine is unique in treating atrophic gastritis. In addition to treating yin and yang deficiency, liver and stomach qi stagnation, cold pathogen invading the stomach, stomach heat excess, food accumulation in gastrointestinal tract, blood stasis blocking collaterals, stomach yin deficiency, spleen and stomach weakness. In modern clinical treatment of gastrointestinal diseases, only the elimination of inflammation and ulcer is the cure index, and inflammation and ulcer are only signs of gastrointestinal diseases. This syndrome is the cause of inflammation and ulcer formation, which is also the main reason why recurrent gastrointestinal diseases cannot be cured and lead to malignant transformation.
In addition to drug treatment, atrophic gastritis should pay more attention to health care and diet conditioning. As the saying goes, the importance of aftercare can be seen from the three treatments and seven aftercare of stomach diseases.
Health care, diet conditioning, fasting to drive away cold, such as cold salad, coke, sprite, carbonated drinks; Eat raw onions, ginger, garlic, leeks, garlic sprouts, peppers, persimmon peppers and other spicy foods; Non-digestible food, such as fried food, sticky food, too greasy food. In particular, patients with atrophic gastritis with hyperplasia and intestinal metaplasia should avoid eating hairtail, potatoes, spinach, milk, pineapple and other foods, do not smoke, and drink strong tea, strong coffee and wine. Keep your mouth light and don't eat food with high salt content. Also pay attention to food hygiene and ban drugs that stimulate the stomach. At the same time, we should maintain a good attitude, relax in everything, don't worry too much, and suffer from too little stomach trouble. Only the correct treatment, the right medicine, and the correct maintenance and conditioning of life can help atrophic gastritis recover as soon as possible.
In short, atrophic gastritis should be treated early, even if you go to the hospital, don't be careless, so as not to miss the treatment opportunity and affect your health.
Drug introduction:
1, ulcer powder
Function: promoting granulation and killing insects, nourishing spleen and stomach.
Indications:
Early-dull pain on an empty stomach. Eat safely and regularly.
Mid-term-2-5 pm, the pain wakes up regularly.
Late stage-severe stomach pain, aggravated after eating and other stimuli, unbearable pain, irregular time, and possible perforation.
Indications: Gastrointestinal ulcers such as gastric ulcer, intestinal ulcer, esophageal ulcer and oral ulcer.
Service mode:
Initial stage-take the bag 30 minutes before each meal.
Moderate qi-take 1 bag before going to bed.
Take 2-3 bags at night.
Remarks: If the pain gets worse after taking the medicine for 2-3 days, it is caused by the reaction with ulcer, which is a normal reflection. The pain will go away in about 2 days. People with severe pain can take less medicine, but they should not stop taking it.
2. Jianwei Granules
Function: promoting blood circulation, removing blood stasis and strengthening spleen and stomach.
Indications: atrophic gastritis, chronic gastritis, superficial gastritis, antritis and reflux gastritis. Diseases caused by low spleen and stomach function, indigestion and malnutrition.
Indications: edema and congestion of digestive tract; Noisy stomach, pain and discomfort; Abdominal distension after meals; Loose or dry stools; Small and anorexic; The elderly have poor appetite and weak body.
Administration method: Take 1 bag after each meal, and add 1-2 bags for heavier ones.
Remarks: If diarrhea gets worse after taking the medicine, it belongs to spleen deficiency and intestinal dampness, which is a normal reflection. It can be reduced according to the severity of symptoms, and it is not necessary to stop taking the medicine. When you have indigestion or loss of appetite, this medicine should be taken for 3-5 days. It can prevent inflammation.
3. Yiqihewei Granules
Function: benefiting qi and regulating stomach, stopping adverse reactions, and being lenient.
Indications: Stomach disease with liver-qi inversion. Such as atrophic gastritis, antritis and reflux gastritis.
Indications: swelling and pain in stomach and hypochondriac ribs, heartburn, poor abdominal gas, burping, dry mouth and bitter taste, dreaminess or insomnia, etc.
Administration method: Take 1 bag after each meal, and add 1-2 bags for heavier ones.
Remarks: It is a normal reaction to aggravate abdominal distension after taking medicine. If you take it in a visible amount, people with gastroptosis must take Jianzhong Shengyang powder first to strengthen the function of the spleen and stomach, and then take this pharmacological gas. Use with caution for patients with mild gastroptosis. Severe cases should not be taken. Take 1 immediately after getting angry to prevent liver depression.
4. Bupi Yichang Powder
Functions: invigorating spleen, warming middle warmer, eliminating dampness and stopping diarrhea.
Indications: acute and chronic enteritis and colitis.
Indications: spleen-yang deficiency, diarrhea and bowel sounds.
Usage: Pay one before each meal.
5. Jianzhong Shengyang Powder
Function: ascending and sinking, invigorating middle energizer and benefiting qi.
Indications: Depression caused by gastroptosis and qi deficiency of middle energizer.
Indications: Postprandial abdominal pain, abdominal distension and weakness of limbs.
Usage: Take one dose on an empty stomach before meals.
6. Sanxiang Wei Qing Powder
Function: clear away stagnation and turbidity, remove filth and bring fragrance.
Indications: Gastrointestinal syndrome caused by transportation disorder and stagnant urine.
Indications: strong taste or peculiar smell in the mouth.
Usage: Take one pill on an empty stomach before each meal.
7. Gastric acid powder
Function: regulating liver and spleen
Indications: hyperacidity
Indications: pantothenic acid, acid regurgitation, and love to eat alkaline food on weekdays.
Usage: Take one dose after each meal, and increase the dose in severe cases.
8. Jiejing Zhitong Powder
Function: regulating qi, dredging collaterals, relieving spasm and pain.
Indications: Pain caused by gastrointestinal spasm and various stomach diseases.
Indications: Stomach twitching pain, except sudden attack pain, then the number of attacks increased and the pain gradually eased.
Function: regulating qi, dredging collaterals, relieving spasm and pain.
9. Han Wei pills
Function: Warming stomach, dispelling cold and relieving pain.
Indications: When the cold pain is aggravated, the stomach and limbs are usually cold.
Indications: excess cold-sudden attack of stomach pain and severe pain.
Deficiency cold-the pain is slow and dull.
Usage: Take 1 before going to bed.
10, Wenweishu Capsule
Pharmaceutical composition: Radix Codonopsis, Atractylodis Rhizoma, Fructus Crataegi, Radix Astragali, Herba Cistanches, etc.
Functions: strengthening the body resistance, warming and nourishing the stomach, promoting qi circulation and relieving pain, and helping yang to warm the middle warmer.
Indications: It is used to treat epigastric pain, flatulence, belching, anorexia, chills and fatigue caused by chronic atrophic gastritis.
1 1, Yinxu Weitong Granule
Ingredients: Radix Glehniae, Radix Ophiopogonis, Fructus Schisandrae Chinensis, Radix Glycyrrhizae, etc.
Function: nourishing yin, benefiting stomach and relieving pain.
Indications: It is used for dull burning pain in the upper abdomen, dry mouth, anorexia and retching caused by deficiency of stomach yin, and is mainly used for the treatment of chronic superficial gastritis, atrophic gastritis, peptic ulcer and other diseases in clinic.
12, Yangweishu Capsule
Medicine composition: Radix Codonopsis, Rhizoma Polygonati, Radix Scrophulariae, Mume Fructus, Atractylodis Rhizoma, Semen Cuscutae, etc.
Functions: strengthening body resistance, nourishing yin and stomach, regulating middle energizer, promoting qi circulation and promoting digestion.
Indications: used for chronic atrophic gastritis, stomach heat, distending pain, hand and foot fever, dry mouth, bitter taste and loss of appetite caused by chronic gastritis.
13, Xuhan Weitong Granule
Medicine composition: Radix Paeoniae Alba, Zingiberis Rhizoma, Radix Codonopsis, Radix Glycyrrhizae, Fructus Jujubae, etc.
Function: Warming stomach and relieving pain, invigorating spleen and benefiting qi.
Indications: It is used for patients with spleen deficiency, weak stomach, dull pain in epigastric cavity, preference for warm pressure, and severe abdominal pain in case of cold or fasting. Duodenal ulcer, chronic atrophic gastritis and other diseases.
14, Sanjiu Weitai
Medicine composition: Sanjiu, Murraya murraya, Radix Paeoniae Alba, Radix Rehmanniae and Radix Aucklandiae.
Function: diminishing inflammation and relieving pain, regulating qi and invigorating stomach.
Indications: superficial gastritis, erosive gastritis, atrophic gastritis and other types of chronic gastritis.
15, mushroom slices
Pharmaceutical ingredients: Hericium erinaceus.
Function: diminishing inflammation and relieving pain, strengthening body resistance and strengthening the constitution.
Indications: chronic atrophic gastritis, peptic ulcer, gastric cancer, esophageal cancer, etc.
16, weinaian capsule
Pharmaceutical ingredients: Radix Astragali, Radix Notoginseng, artificial bezoar and pearl powder.
Functions: invigorating qi and spleen, calming the heart and calming the nerves, promoting qi and blood circulation, diminishing inflammation and promoting granulation.
Indications: chronic superficial gastritis, atrophic gastritis, gastric and duodenal ulcer.
17, Weikangling Capsule
Medicine composition: Radix Paeoniae Alba, Radix Glycyrrhizae, Rhizoma Corydalis, Radix Notoginseng, etc.
Functions: softening liver and stomach, removing blood stasis, stopping bleeding, relieving pain, removing putrefaction and promoting granulation.
Indications: It is suitable for acute gastritis, chronic superficial gastritis, chronic atrophic gastritis, peptic ulcer and gastrorrhagia.
18, Yangwei Granule
Medicine composition: Radix Astragali, Radix Paeoniae Alba, Rhizoma Dioscoreae, Rhizoma Cyperi, Radix Codonopsis, Radix Glycyrrhizae, Pericarpium Citri Tangerinae, etc.
Function: nourishing stomach and spleen, regulating qi and regulating middle energizer.
Indications: Chronic atrophic gastritis.
19, compound weileshu oral liquid
Pharmaceutical ingredients: Hericium erinaceus concentrated solution, royal jelly, honey, etc.
Function: Benefiting five internal organs, helping digestion and improving immunity.
Indications: It can be used to treat peptic ulcer, gastritis, chronic atrophic gastritis, spleen and stomach weakness and gastrointestinal diseases.