? Under normal circumstances, the signals of exciting and contracting myocardium are sent from the sinoatrial node of the right atrium and transmitted to other parts of the heart in turn. Muscles in all parts of the left and right atria contract rhythmically while the ventricles relax (called sinus rhythm), which promotes the blood in the atria to enter the ventricles and ensures that the blood enters the aorta from the ventricles and reaches the tissues and organs of the whole body. When rheumatic heart disease, coronary heart disease and other reasons make the signals of myocardial excitation and contraction transmit abnormally in the atrium, and repeated reentry makes the muscles of various parts of the atrium beat rapidly and disorderly, and lose coordination and correlation with ventricular beat, it is called atrial fibrillation, which is referred to as atrial fibrillation for short.
? ① Symptomatic: During the onset of atrial fibrillation, in addition to the hemodynamic changes caused by basic heart disease, atrial contraction function is lost, ventricular contraction becomes irregular, and ventricular rate increases. The most common symptom of patients is palpitation. If combined with coronary heart disease, patients may have angina pectoris, dizziness, syncope, and severe heart failure and shock. Patients with rheumatic mitral stenosis often induce acute pulmonary edema, and patients with pulmonary hypertension may have hemoptysis. ?
? ② Asymptomatic: Some patients with chronic and moderate atrial fibrillation can be asymptomatic, especially the elderly, which are often found in physical examination or electrocardiogram. ?
? ③ Atypical symptoms: seen in chronic or moderate atrial fibrillation, the patient has no palpitation, but may have fatigue, precordial discomfort or slight pain, and the diagnosis can only be made after further examination.
The above content comes from Baidu Encyclopedia.
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