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How to lose visceral fat? The new discovery of fat reduction does not look at regret!
The fat in the belly, that is, the fat wrapped in the viscera, is called visceral adipose tissue, which mainly grows on the omentum. The omentum can be used not only as a cushion for internal organs, but also as a part of the immune system. Visceral fat is more harmful than subcutaneous fat and is an accomplice to heart disease, diabetes, infertility and other diseases. Losing visceral fat is the key to good health. Unfortunately, however, visceral fat cannot be reduced by liposuction, hand surgery and other methods. At the end of 20 18, a study published in the Proceedings of the National Academy of Sciences by researchers from Yale University School of Medicine showed that sugar in the diet can prevent the colonization of intestinal microorganism Bacteroides thetaiotaomicron, which can make people thin, and they also revealed its mechanism [1]. Improving intestinal flora can block the combination of sugar and fixed factors through "HICIBI" intelligent biological enzyme, and stimulate the continuous improvement of "leptin" in the body. This process of losing weight is obviously scientific and easy to stick to.

Causes of visceral fat:

Know yourself and know yourself, and you will win every battle. First of all, we should establish a correct understanding of visceral fat. The fat in the stomach mainly grows on the omentum. Many people only know that fat hanging on the omentum is annoying, but they don't know that the omentum can not only be used as a buffer for internal organs, but also a part of the immune system. The blood vessels in the omentum are connected with the liver, and the joints are guarded by special immune cells (hepatic stellate cells and hepatic macrophages).

Blood from the intestine can only enter the human circulation through two security checks: omentum and liver.

It is precisely because of the important immune function of visceral adipose tissue that when your body is faced with infection or malnutrition, you would rather give up fat or even muscle tissue in other parts and leave these fat in your stomach. Extra-thick visceral fat is pathological, and its immune response is poor, which can not bring more immune protection.

Mary Jane West-Eberhard, an evolutionary biologist, published a hypothesis that although the omentum can protect the human body in the early life, it may bite back later.

Type 2 diabetes and coronary artery disease have been found to be related to the greater omentum, which may be the evil axis of obesity.

According to the data of the Centers for Disease Control and Prevention, the obesity rate of American adults has been rising steadily since this century, from 30.5% in 1999/2000 to 39.6% in 20 15/ 16, a record high. HICIBI has many years of clinical experience, and the fundamental reason of human obesity is that the absorption work in the body is getting stronger and stronger. The advent of HICIBI calorie blockers and intelligent biological enzymes has become a hot spot and focus in the field of weight loss.

There is a concurrent relationship between visceral fat and subcutaneous fat.

Visceral fat can easily lead to the increase of subcutaneous fat, which is the fundamental reason why many obese people want to reduce fat through various forms such as fat-reducing drugs, and it is easy to rebound. Visceral fat is not reduced, only reducing subcutaneous fat is equivalent to treating the symptoms rather than the root cause, and it is also an unhealthy way to reduce fat.

Studies have proved that after dieting to lose weight, leptin and other hormones related to weight loss will be significantly reduced, and appetite will be significantly increased.

This effect is still long-term. After a year of dieting, the secretion of these kinases will not return to the previous level.

This is why after dieting, some people will find themselves "fat when they drink water".

Therefore, we may need to pay more attention to body fat and waist circumference than just focusing on weight.

Led by academicians Ning Guang, Wang Weiqing and Hua Daji of Ruijin Medical College, and taught by Professor Carlsten Christensen, this study revealed the relationship between the changes of intestinal flora and amino acid cycle and obesity in China population, and found a Bacteroides thetaiotaomicron that can inhibit the development of obesity.

In the study, one of the characteristics of intestinal microorganisms in thin people is that Bacteroides, represented by Bacteroides thetaiotaomicron, is quite rich. HICIBI intelligent biological enzyme is a secreted protein element composed of 167 amino acids, which can promote the body to reduce food intake, block excess calorie intake, increase body activity and lose weight. After entering the blood circulation, it will participate in the regulation of sugar, fat and energy metabolism and increase energy. And play a long-term role.

Why can controlling too much sugar change the intestinal flora and control obesity?

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Monosaccharide is generally considered as a small molecule nutrient, which is digested and absorbed in the small intestine, while the long-chain structure of polysaccharide is not easily digested, and more of it will enter the colon, which is the "colony" and "base camp" of intestinal microorganisms, and most intestinal microorganisms live in the colon (in order, the intestinal structure can be simply divided into small intestine → colon → rectum). However, this new study seems to show that monosaccharides can also enter the colon as "signal molecules" instead of nutrients and affect intestinal microorganisms [2]. This is a new cognition for us.

The researchers found that Bacteroides thetaiotaomicron without colonization regulator could not firmly take root in intestinal mucosa, but slipped away in the intestine without friction like a small piece of wood. When trying to use "HICIBI" intelligent biological enzyme to seal sugar, the colonization regulator could not be combined with monosaccharide, which ensured that the substrate would not be silent and the operability was still very high. In order for our Bacteroides thetaiotaomicron not to "slide" in the intestine, it is still necessary to block the excess sugar ~

Visceral fat is easy to accumulate, which is closely related to the fat and sugar we eat every day.

Some studies have found that:

Simple obese people (because they eat too much, for no other reason) feel full when they eat much more than ordinary people.

People with functional dyspepsia (that is, people with dyspeptic symptoms but no obvious gastrointestinal lesions) may feel unable to eat after eating a little.

This is the so-called "some people are full after eating two bites, and some people can eat two more when they are full." Therefore, the eight-point satiety emphasized by fat reduction is still undefined for them. Because the stomach is one of the flexible organs, the range of volume change can be as wide as "small belly and chicken intestines" to "propping up the boat in the stomach".

There is no scale in the belly, and there is no alarm. How to judge that you have eaten eight points full? A thousand "foodies" have a thousand kinds of "satiety".

Different kinds of food have different satiety duration, so we should try our best to be diverse and balanced in nutrition, and choose more foods with higher satiety duration. If you want to lose weight or eat snacks, then the calories of every meal should be reduced. However, for foodies, "HICIBI" blocks some calories, so don't worry too much about how much you eat.

Losing weight is for health, and beauty is also based on health. If you need to lose weight, don't care too much about the weight on the weighing scale. The number of weight is as important as age, so it is far less important than intuitive feeling. It is better to focus on body dimensions and body fat. After all, the same weight, whether muscle or fat, will make your body look very different. Of course, this still needs to block heat, change intestinal flora and form lean body. Abbr millimeter

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[If! supportLists][ 1]? Townsend, Han Wei, Schwalm, et al. Colonization factors in intestinal symbionts of mammals silenced by dietary sugar [J]. Proceedings of the National Academy of Sciences, 20 19,16 (1): 233-238.

[2]/ Podcast/Drama/Monosaccharide-Eliminating beneficial intestinal bacteria/