How do thin people practice? Have you heard of it there? How to lose weight? How can thin people never get fat? This is normal. If you want to exercise and become thin, you need to start with diet. Experts say it won't make you fat. Let's have a look. I hope the skinny people below can help you practice!
How do thin people practice? 1 thin people eat open breakfast.
Up to 96% people eat breakfast. The most common foods include fruit (565,438+0%), dairy products (465,438+0%), cold cereal or cereal (33%), and about 30% people eat bread, eggs and hot cereal.
Lunch and dinner.
More than13 of the respondents eat salad for lunch and vegetables for dinner every day. Most people's favorite meat is chicken with low fat content.
dessert
Don't eat cakes and biscuits as afternoon tea. The most common snack these respondents eat is fruit! The second most common food is nuts.
Drinks
Nearly 70% people here never drink soft drinks (such as soda), or only drink sugar-free versions, and only 10% people drink cola. In addition, nearly 20% people don't drink.
sports
Nearly 40% of the respondents exercise 1 to 3 days a week, and only 10% do not exercise at all.
Eat a balanced diet and eat more healthy food.
The researchers said that this study showed an important message that slim people almost always eat breakfast, which echoed previous studies that emphasized the importance of breakfast. Moreover, these people not only eat breakfast regularly, but also choose healthy foods such as fruits and vegetables. The study was published in Obesity Week 20 15 in Los Angeles, USA.
In addition, nearly half of the respondents in this study did not go on a diet, and even more than 1% said that they were not losing weight at all! Many people don't eat staple food or breakfast in order to lose weight, but only three normal meals, balanced nutrition and healthy ingredients can maintain normal metabolism and help them eat a slim figure, not just reduce calories!
How do thin people practice? Background: Down-regulation of adiponectin level is regulated by genetic and environmental factors, which will damage adipose tissue function and increase cardiovascular risk. At the same time, different dietary interventions have potential effects on regulating adiponectin secretion. The purpose of this study is to explore the modification effects of childhood dietary patterns on adiponectin-related genes that affect metabolic health phenotype, and whether these effects are different between normal weight and overweight and obesity.
Methods: This study is a large prospective cohort study with long-term follow-up from children to adults. Baseline analysis included 3482 children aged 6~ 18 years from Beijing Children and Adolescents Metabolic Syndrome Cohort Study (BCAMS), some of whom had completed the in-depth follow-up of 10 years. Metabolic abnormality is defined as insulin resistance index greater than or equal to 3.0, or any component of metabolic syndrome.
Results: Multivariate regression analysis showed that one SNP locus of adiponectin gene (or =1.25,95% CI:1.08 ~1.46, P = 0.004) and one SNP locus of adiponectin receptor gene CDH 13. SNP reduces the G locus of adiponectin, which is related to the higher metabolic risk of normal children with poor dietary patterns, but the correlation between G locus and metabolic risk disappears in a good dietary pattern. The subgroup analysis of ten-year follow-up also verified this interaction.
Conclusion: For the first time, we found that the interaction between genes and nutrition changed the susceptibility of children with abnormal metabolism in China, especially those with normal weight. Individualized nutrition scheme design of genotyping is a potential early preventive measure to effectively improve metabolic health.
Early developmental environment and gene interaction predict the phenotype of metabolic obesity with normal weight in childhood: the BCAMS study.
Background: Obesity is an independent risk factor for many metabolic disorders such as type 2 diabetes, hypertension and dyslipidemia. However, despite normal weight, a considerable proportion of people still have obesity-related metabolic abnormalities, that is, normal weight metabolic obesity (MONW). There is a lack of research on this metabolic phenotype, especially in children and adolescents. The purpose of this study is to explore the early developmental environment and genetic susceptibility factors related to the occurrence and development of MONW phenotype in a large cohort of children and adolescents.
Methods: This study was included in the Beijing Children and Adolescent Metabolic Syndrome Cohort Study (BCAMS) 1484 normal-weight children aged 6~ 18 years, of whom 588 had metabolic abnormalities. Evaluate and record the birth weight, childhood lifestyle and social and environmental factors of each subject. Complete the typing detection of body mass index and genes related to glucose metabolism in 20 GWAS studies of East Asian population. Metabolic obesity is defined as any component with insulin resistance index greater than or equal to 3.0 or with/without metabolic syndrome.
Results: Low birth weight, increased body mass index, decreased daily exercise time, low education level of mother and low family income were independent risk factors for MONW (P; 0. 1)。 In addition, the correlation between higher birth weight and CDKAL 1- MONW can be further amplified by the overall environmental model of childhood (comprehensive effect of active exercise, higher education level of mothers and family income) (Pinteraction=0.0 17).
Conclusion: Early developmental environment (especially intrauterine developmental environment), genetic susceptibility and their interaction are related to the MONW phenotype in childhood. Our research provides a new understanding of the occurrence and development mechanism of MONW, and also provides a basis for long-term prevention and treatment strategies from intrauterine to children. This prevention and control strategy is especially suitable for people with specific genetic susceptibility.