The next step of arteriosclerosis is plaque, then embolism, and it is not too late to exercise again.

The most important thing is to control diet, reduce the intake of high-fat and high-cholesterol foods and lose weight.

Because of arteriosclerosis, blood circulation pressure is high, so exercise should be gradual, pay attention to your body voice, and gradually add a lot. When you have palpitation, shortness of breath, dizziness and nausea, you must rest and adjust in time, and prepare some commonly used drugs when necessary.

In addition, if you exercise on an empty stomach, you should bring sports drinks and supplement sugar, salt and electrolyte to prevent hypoglycemia. You can also tell the staff to pay special attention.

It's safer to go to the gym than to faint outside. Just don't kill yourself.

Can atherosclerosis climb a mountain? Many people like to climb mountains as a kind of exercise to prevent diseases, keep fit and lose weight. These are all fine. But please don't forget that mountain climbing is a sport that consumes a lot of oxygen, and not everyone is suitable for it. So, who are not suitable for mountain climbing? Let's take a look.

1. Young people with cardiomyopathy or rheumatic heart disease are not suitable for mountain climbing. Young people with sudden death have dilated or hypertrophic cardiomyopathy, so patients with the above two diseases should not climb mountains.

2. Elderly people with poor health and patients with cardiovascular and cerebrovascular diseases should not climb mountains. The functions of various organs of the elderly are declining, and most of them suffer from some chronic diseases to varying degrees. Mountain climbing is a sport that consumes a lot of oxygen. Old people who are going to climb mountains must do some less intense exercise first so as to have an adaptation process. People of a certain age (women over 50 years old and men over 40 years old) belong to the high-risk group of coronary heart disease. People who already have hypertension and coronary heart disease, especially those with chronic coronary insufficiency, are not suitable for mountain climbing.

3. Some patients with chronic diseases should not climb mountains. No matter what age, patients with heart disease and hypertension should not climb mountains, and some patients with chronic diseases should not climb mountains. These chronic diseases include arthralgia, chronic nephritis, nephropathy, hematopathy, chronic tracheitis, cor pulmonale, diabetes with complications, gout, lupus erythematosus, dermatomyositis and other rheumatic diseases, cirrhosis and so on. Patients with chronic diseases should climb slowly even if they climb mountains. Don't force them to climb to the top of the mountain.

Middle-aged people who often don't exercise shouldn't suddenly take part in large-scale mountaineering activities. Middle-aged people are an extraordinary group, shouldering two heavy burdens of work and family, sandwiched between the elderly and children. Middle-aged people are busy with their careers, but their physical fitness, psychological endurance, immunity and endocrine function are declining day by day. Besides, they are very busy and have no time to take care of their health. They are prone to various diseases, and early detection and treatment are not easy. I seldom exercise at ordinary times, and suddenly go climbing, realizing that I am more flexible, stronger and often climb faster than the elderly. Activities surge, physical load suddenly increases, and as a result, heart and lung diseases are easily induced.

In the process of climbing the mountain, if you have sudden symptoms, you must take necessary measures to save yourself. For example, if you feel suffocated in the precordial area, don't keep climbing, but lie flat in a relatively comfortable place to rest. At this time, you should immediately loosen your belt and tie and tilt your head to one side to avoid suffocation caused by vomit. Pay attention to keep warm, because cold will aggravate vasoconstriction and platelet aggregation. Young and middle-aged people should have regular physical examinations and find potential hidden dangers of heart and lung as soon as possible. If you don't sleep well at night after climbing the mountain, it means that you exercise too much and need to adjust your exercise.

Can atherosclerosis be reversed? The world is so wonderful. Medical scientists are constantly denying the health care methods they take for granted through scientific research, and at the same time, they are also discovering the hope of treating irreversible diseases. As Professor Hong Zhaoguang said, God makes you sick, and at the same time, God spreads drugs to treat diseases in nature, waiting for you to discover. Atherosclerosis is the main cause of coronary heart disease. In the past, people thought this hardening was irreversible, but now it can be reversed. This hope can save many people from myocardial infarction, so as to stay away from heart bypass and heart stent surgery!

According to the latest report in The Times, a landmark study shows that millions of heart patients can take higher doses of cholesterol-lowering drugs to reverse their illness and effectively avoid heart attacks. This is the first time that scientists have found a way to reverse the chronic deposition of arterial fat. This medical progress is also called the "holy grail" to prevent coronary heart disease.

Atherosclerosis leads to myocardial infarction and stroke.

This study found that atherosclerosis in patients with coronary heart disease was significantly improved by taking higher doses of statins. Atherosclerosis is a form of arteriosclerosis, which is characterized by the deposition of atherosclerotic blocks containing cholesterol and fat in the innermost layer of arterial wall. This process is often compared to the scale formation process of water pipes, which will lead to narrower and narrower vascular passages and fatal diseases including heart attacks and strokes. Prior to this, scientists focused on slowing down the process of this irreversible disease. However, an important study involving rosuvastatin shows that the coronary artery deterioration of patients can indeed be improved after taking a large amount of this drug. British scientists engaged in this research said at the annual meeting of the College of Cardiology held in the United States that taking a large number of cholesterol-lowering statins may halve the risk of heart attack.

Atherosclerosis is hopeful to be reversed.

Neil Ullen, a cardiologist at Edinburgh Royal Hospital, said that for those patients with clinical response, this method may greatly prolong their lives. The Institute is the only British research center involved in this research. Ulun said: "In the past, people always thought that once you unfortunately got arteriosclerosis, it meant that there was no cure for you. Now our research shows that this disease can actually be significantly alleviated. " In Britain, it is estimated that there are at least 2 million patients with arteriosclerosis. Although some people are prone to this disease due to genetic reasons, this disease can be prevented to a great extent if people strictly follow a healthy lifestyle. In the past two years, 53 research centers in the United States, Canada, Europe and Australia have studied 349 heart patients and found that statins are very effective in lowering cholesterol.

In recent years, other studies have shown that atherosclerosis can subside. Animal experiments have proved that feeding rabbits with high cholesterol diet can cause experimental atherosclerosis, and once the experimental diet is stopped, the disease will gradually subside. Similar results have been obtained in monkeys that are closer to humans. Epidemiological investigation found that after the first century war, the incidence of atherosclerosis in some countries decreased significantly. After the second century war, with the decrease of butter and fat intake, the mortality of cardiovascular diseases in Finland, Norway and Sweden decreased significantly. This shows that changing lifestyle can reduce atherosclerotic plaques and reverse atherosclerosis.

Hepatitis C and early cirrhosis, can you go to the gym for fitness? number

After liver cirrhosis, portal hypertension, low stomach and esophageal varices, splenomegaly. Doing exercise is very prone to accidents, and it is also easy to lead to the accelerated development of the disease.

Just do moderate activities at ordinary times.

Hello, cerebral atherosclerosis, but poor blood circulation will only cause arteriosclerosis and even cerebral hemorrhage. You can take some decoction for invigorating qi, promoting blood circulation and removing blood stasis.

Can atherosclerosis be reversed? Atherosclerosis can be reversed, which is manifested in the removal of cholesterol-tired foam macrophages from the intima of arteries. Recently, an experimental study conducted by Park and his colleagues showed that these foam-like cells were influenced by the interaction of oxidized low-density lipoprotein (LDL) and could migrate again after being exposed to important antioxidants, such as resveratrol, a polyphenol compound found in grapes and red wine, which has potential anti-atherosclerosis ability. Atherosclerosis was once considered as a fat storage disease-a lesion or plaque that leads to arterial obstruction-which originated from the accumulation of fat in arteries. Now it is considered that the nature of atherosclerotic injury is more complicated. It is characterized by local inflammation of the vascular wall, and there are many signs of chronic inflammation in the local area, including the existence of macrophages, dendritic cells and lymphocytes. These white blood cells gather in specific places, and the endothelial cells below them show activated or inflammatory phenotype. At first, the lesion showed liposuction, and under the dual influence of genes and lifestyle, it developed towards pathological injury. However, the vast majority of plaques are the result of years of progress in asymmetric diseases, and the final blocking events often appear in the form of sudden embolism. During the whole life cycle of the disease, from lipid streaks to the final disease formation, many events may occur to accelerate, delay or even reverse the progress of the disease. Chronic inflammation of arterial wall, as the main part of atherosclerotic lesions, raises the question of the relationship between inflammation and disease degree. Inflammation is the normal response of the body to trauma or infection to keep the body stable. Usually, it is self-limiting and restores the stability of the tissue. Chronic inflammation occurs when the inflammatory response cannot solve the events that cause inflammation or the self-limiting mechanism is destroyed. Some factors that promote atherosclerosis include hyperlipidemia, lipid oxidation, the accumulation of white blood cells in arterial wall and the formation of foam-like cells derived from macrophages (Figure 1). The production of oxidized lipid with biological activity in vivo can trigger and regulate this inflammatory cell event. The accumulation of cholesterol (in the form of macrophage-derived foam cells) in local lesions is considered to be caused by the discharge of key lipids, the decrease of catalytic mechanism activity or both. At the molecular level, scavenger receptors on the surface of macrophages play a key role in the formation of foam cells. For example, CD36 mediates the uptake of oxidized LDL by recognizing its special truncated fatty acid moiety and oxidized phosphatidylcholine. Consistent with this observation, it was found that CD36 was lacking in hyperlipidemia mice resistant to atherosclerosis. Bioactive lipids of oxidized LDL that can be internalized by CD36 can activate nuclear receptors, such as peroxisome proliferator-activated receptor γ, thus starting the transcription process of up-regulating the expression of CD36 and other genes related to lipid metabolism. CD36 is also a direct signal molecule; It started the key cascade signal. In their recent research, Park et al. described a new function of CD36: regulator of cell migration. Macrophages participate in the intima, which leads to the progression of the lesion. Removing macrophages from the intima will promote healing (Figure 1). The movement of macrophages includes the formation of flaky pseudopodia (the protrusion of cytoskeleton composed of actin in cells), the rupture of local adhesive connections and the formation of new connections. These processes in turn depend on the dynamic control of actin skeleton and local adhesion. Park et al. found that the signal of CD36 caused by oxidized LDL (non-LDL) promoted the polymerization and "firm" adhesion of actin, so foam-like macrophages were confined to the lesion site. They found that CD36 signal activates the process of cell colonization by producing reactive oxygen species (ROS), which indirectly activates local adhesion kinase, leading to the increase of actin polymerization level. The imbalance between reactive oxygen species (ROS) and antioxidant synthesis is described as oxidation pressure. The production of ROS in the body is offset by several antioxidant mechanisms. The first mechanism is the production process itself. NADPH oxidase is the enzyme responsible for ROS production, and inhibiting this enzyme provides an attractive direction for the treatment of many diseases. The second direction is antioxidants, although one effect is sometimes not enough to deal with ROS. Park et al. used therapeutic doses of two oxidase inhibitors, apocynin and diphenyliodonium, and antioxidants, N- acetylcysteine and resveratrol, to inhibit ROS produced by macrophages in vitro. In the presence of oxidized low density lipoprotein, the cell migration ability is then restored, which makes the foam cells get rid of local adhesion, which is a necessary condition for cell diffusion. Antioxidants (including other antioxidants) used by Park et al. can provide therapeutic strategies for atherosclerosis by reducing the adhesion and diffusion of macrophage-derived foam cells in intima. The reversal of the non-migration characteristics of macrophages will make foam cells have the ability to act, which is necessary for cell expulsion and damage reduction. Interested comrades discuss atherosclerosis together. Surgery can't prevent vein graft from hardening again, so this disease may need a breakthrough in basic science.

Is coronary atherosclerotic plaque the same as coronary atherosclerosis? It's actually a concept. First there is coronary atherosclerosis, then there is coronary atherosclerotic plaque formation, and finally it develops into coronary heart disease.

Atherosclerosis of the main artery Atherosclerosis refers to the fact that after the limited injury of the intima in the early stage, the lipids in the blood will deposit on the intima, and then the connective tissue of the intima will proliferate, so that the intima will locally thicken or swell, forming plaques, and then necrosis, collapse and softening will occur under many plaques, which looks like a layer of rice porridge on the surface of the intima, so it is called atherosclerosis. This kind of hardened plaque is localized and narrow, which blocks blood vessels to varying degrees, affects the smoothness of blood flow, and leads to ischemia and dysfunction of corresponding organs in the body. The most common sites of atherosclerosis are coronary artery and cerebral artery, followed by renal artery, abdominal aorta and lower extremity artery.

Atherosclerosis is one of the most clinically significant arterial diseases and poses the greatest threat to human beings. With the improvement of people's living standards in China, the incidence of atherosclerosis is getting higher and higher, which is enough to attract people's attention and prevention.

abstract

Atherosclerosis is the most common and important type of atherosclerosis. This disease mainly involves large and medium-sized muscle elastic arteries, especially aorta, coronary artery and cerebral artery, which often leads to serious consequences such as lumen occlusion or wall rupture and bleeding.

Atherosclerosis is more common in men over 40 years old and postmenopausal women. This disease is often accompanied by hypertension, hypercholesterolemia or diabetes. Mental workers are common and do great harm to people's health, which is one of the main causes of death of the elderly.

The cause of disease

First, hypertension: the incidence of atherosclerosis in patients with hypertension has increased significantly.

Second, hyperlipidemia: atherosclerosis is common in hypercholesterolemia.

Third, smoking.

Fourth, diabetes.

Fifth, obesity.

symptom

This mainly depends on vascular diseases and the degree of ischemia of the affected organs. Atherosclerosis of major arteries is usually asymptomatic. If the diameter of coronary atherosclerosis exceeds 75%, angina pectoris, myocardial infarction, arrhythmia and even sudden death may occur. Cerebral arteriosclerosis can cause cerebral ischemia, brain atrophy, or cerebral vascular rupture and bleeding. Renal atherosclerosis often causes nocturia, refractory hypertension and severe renal insufficiency. Mesenteric atherosclerosis can be manifested as abdominal pain and bloody stool after a full meal. Patients with severe stenosis of vascular lumen caused by atherosclerosis of lower limbs may have intermittent claudication, pulse loss of dorsal artery of foot, and even gangrene in severe cases.

cheque

1. Patients usually have increased blood cholesterol and triglycerides, decreased high-density lipoprotein and abnormal lipoprotein electrophoresis patterns. Most patients showed type ⅲ or ⅳ hyperlipoproteinemia.

2.x-ray examination shows that the aorta is elongated, dilated, twisted and sometimes calcified.

3. Arteriography can show the lumen stenosis, lesion location and scope of limb artery, renal artery and coronary artery caused by atherosclerosis.

4. Doppler ultrasound examination is helpful to judge the patency of arterial and renal blood flow in limbs.

treat cordially

First, the general treatment:

(1) Reasonable diet: The total calories in the diet should not be too high to prevent overweight.

(2) Insist on moderate physical activity.

(three) reasonable arrangements for work and life.

(4) Others: advocate non-smoking and drink a little.

Second, control the susceptible factors: If you have diabetes, you should control your blood sugar in time, including diet control. Hypoglycemic drugs for the treatment of type 2 diabetes should not cause hyperinsulinemia, such as damekang; If there is hypertension, antihypertensive drugs should be given to reduce blood pressure to an appropriate level. If there is an increase in blood cholesterol, high cholesterol should be controlled and lipid-lowering drugs should be given appropriately.

2. Hypolipidemic drugs: 1, cholestyramine. 2. antonin. 3. nicotinic acid. 4. Unsaturated fatty acids. Such as Yishouning, Xuezhiping, Xinmaile, etc. 6. Polysaccharide sodium sulfate.

Third, antiplatelet drugs:

1, aspirin.

2. Pan Shengding.

3. tikrit.

Can atherosclerosis be restored? Can atherosclerosis eat longan water and eggs? Hello, this does not affect atherosclerosis.